To Aai - Baba ii
نویسنده
چکیده
SULE, AMBARISH MUKUND Hardware-Software Codesign of a Programmable Wireless Receiver System-on-a-chip. (Under the direction of Prof. William Rhett Davis). With gate counts and system complexity growing rapidly, engineers have to find efficient ways of designing hardware circuits. The advent of Hardware Description Languages and synthesis methodologies improved designer productivity by raising the abstraction level. With advances in semiconductor manufacturing technology, however, there is still a growing productivity gap between the number of transistorsper-chip that can be fabricated and the transistors-per-day that can be effectively designed[13]. Increasing costs of design encourage reusing cores. Various kinds of Intellectual Property(IP) cores are now widely available and are used in making Integrated Circuits(IC). These System-on-a-chip(SOC) ICs generally contain a microprocessor as one of their IP cores in order to make them more flexible. This heterogeneity of hardware has increased challenges in verification. It is widely estimated that between 60%–80% of the design effort is dedicated to verification[12] with almost half of that time spent in construction and debugging of the simulation environments. Unfortunately, the high costs of industrial IP have made it difficult to explore SOC verification at Universities. This thesis describes the building of a Programmable Wireless Receiver SOC using hardware-software codesign techniques. The SOC is comprised of a general purpose Central Processing Unit(CPU) and a baseband coprocessor with some glue logic. The CPU used is open-source, making it appropriate for teaching SOC verification as part of a university curriculum. The simulation environment adopted to verify the system and its documentation is an important product of this thesis. The thesis can be used as a guideline for designing CPU-based SOCs. Hardware-Software Codesign of a Programmable Wireless Receiver System-on-a-chip
منابع مشابه
Aristolochic Acid I Induced Autophagy Extenuates Cell Apoptosis via ERK 1/2 Pathway in Renal Tubular Epithelial Cells
Autophagy is a lysosomal degradation pathway that is essential for cell survival and tissue homeostasis. However, limited information is available about autophagy in aristolochic acid (AA) nephropathy. In this study, we investigated the role of autophagy and related signaling pathway during progression of AAI-induced injury to renal tubular epithelial cells (NRK52E cells). The results showed th...
متن کاملA Real-time, Event Based Driver Alert System for Accident Avoidance Due to Red Light Running
To my family for their immense love and support And Varun for believing in me ACKNOWLEDGEMENTS I would first of all like to thank Prof. Aniruddha Gokhale for showing faith in my abilities throughout my stay at Vanderbilt. His research thought process and teaching skills always influenced me. I would also like to thank Dr. Jules White for his useful comments and support for my thesis. encouragem...
متن کاملHuman cytosolic enzymes involved in the metabolic activation of carcinogenic aristolochic acid: evidence for reductive activation by human NAD(P)H:quinone oxidoreductase.
Aristolochic acid (AA), a naturally occurring nephrotoxin and carcinogen, has been associated with the development of urothelial cancer in humans. Understanding which human enzymes are involved in AA metabolism is important in the assessment of an individual's susceptibility to this carcinogen. Using the 32P-postlabeling assay we examined the ability of enzymes of cytosolic samples from 10 diff...
متن کاملUsing polymerase arrest to detect DNA binding specificity of aristolochic acid in the mouse H-ras gene.
The distribution of DNA adducts formed by the two main components, aristolochic acid I (AAI) and aristolochic acid II (AAII), of the carcinogenic plant extract aristolochic acid (AA) was examined in a plasmid containing exon 2 of the mouse c-H-ras gene by a polymerase arrest assay. AAI and AAII were reacted with plasmid DNA by reductive activation and the resulting DNA adducts were identified a...
متن کاملCutting edge: intravenous Ig inhibits invariant NKT cell-mediated allergic airway inflammation through FcγRIIIA-dependent mechanisms.
Despite their increasing use in autoimmune, inflammatory, and allergic conditions, the mechanism of action of i.v. Igs (IVIg) is poorly understood. On the basis of the critical role of invariant NKT (iNKT) cells in allergic airway inflammation (AAI) and their constitutive expression of the low-affinity IgG receptor FcγRIIIA, we surmised that IVIg targets iNKT cells to exert their anti-inflammat...
متن کاملComparison of the oxidation of carcinogenic aristolochic acid I and II by microsomal cytochromes P450 in vitro: experimental and theoretical approaches
Abstract The herbal drug aristolochic acid, a natural mixture of 8-methoxy-6-nitrophenanthro[3,4-d]-1,3-dioxole-5-carboxylic acid (AAI) and 6-nitrophenanthro[3,4-d]-1,3-dioxole-5-carboxylic acid (AAII), is derived from Aristolochia species and is the cause of two nephropathies. Ingestion of aristolochic acid is associated with the development of urothelial tumors linked with aristolochic acid n...
متن کاملذخیره در منابع من
با ذخیره ی این منبع در منابع من، دسترسی به آن را برای استفاده های بعدی آسان تر کنید
عنوان ژورنال:
دوره شماره
صفحات -
تاریخ انتشار 2003